Table 1.

Contributory causes of ESRD in young patients (n=2304, 1998–2009)

Contributory Causes of ESRDFemale (n=1049)Male (n=1255)Total (N=2304)
Glomerulonephropathy
 Unknown GN486 (46.3)675 (53.8)1161 (50.4)
 Systemic lupus erythematous164 (15.6)57 (4.5)221 (9.6)
 Henoch-Schönlein purpura9 (0.9)3 (0.2)12 (0.5)
 Scleroderma1 (0.1)1 (0.1)2 (0.1)
 Hemolytic-uremic syndrome2 (0.2)3 (0.2)5 (0.2)
 Wegener granulomatosis2 (0.2)2 (0.1)
 Goodpasture syndrome7 (0.7)2 (0.2)9 (0.4)
Genetic and metabolic diseases
 Type 1 diabetes mellitus100 (9.5)106 (8.4)206 (8.9)
 Amino-acid transport disorders1 (0.1)1 (0.0)
 Glycogen storage diseases1 (0.1)1 (0.0)
 Mucopolysaccharidosis1 (0.1)1 (0.0)
 Metabolic disorders7 (0.7)7 (0.6)14 (0.6)
 Lysosomal storage disorders1 (0.1)3 (0.2)4 (0.2)
 Tuberous sclerosis3 (0.3)1 (0.1)4 (0.2)
 Amyloidosis1 (0.1)1 (0.1)2 (0.1)
Congenital anomalies (CAKUT)
 Renal hypoplasia, dysplasia, oligonephronia18 (1.7)26 (2.1)44 (1.9)
 Cystic kidney disease9 (0.9)27 (2.2)36 (1.6)
 Congenital anomalies of urinary system2 (0.2)2 (0.2)4 (0.2)
 Specified anomalies of kidney8 (0.8)13 (1.0)21 (0.9)
Renal and urinary tract tumor19 (1.8)17 (1.4)36 (1.6)
Nephrolithiasis7 (0.7)23 (1.8)30 (1.3)
Renal trauma1 (0.1)4 (0.3)5 (0.2)
ESRD with hypertension203 (19.4)280 (22.3)483 (21.0)
  • Values are expressed as number (percentage) of patients. CAKUT, congenital anomalies of the kidney and urinary tract.