Article Information
PubMed
Published By
Print ISSN
Online ISSN
History
- Published online July 6, 2009.
Copyright & Usage
Copyright © 2009 by the American Society of Nephrology
Author Information
- Mitsuo Kato,
- Laura Arce,
- Rama Natarajan
- Correspondence:
Dr. Rama Natarajan, Gonda Diabetes Center Beckman Research Institute of the City of Hope 1500 East Duarte Road, Duarte, CA 91010. Phone: 626-256-4673, ext. 62289; Fax: 626-301-8136; E-mail: rnatarajan{at}coh.org or Dr. Mitsuo Kato, Phone: 626-256-4673, ext. 63996; Fax: 626-301-8136; E-mail: mkato{at}coh.org
Statistics from Altmetric.com
Article usage
Cited By...
In this issue
MicroRNAs and Their Role in Progressive Kidney Diseases
Jump to section
- Article
- Abstract
- Biogenesis and Mechanism of Action of miRs
- miRs in Diabetes and Metabolism
- Diabetic Nephropathy and TGF-β Actions
- miRs in the Kidney: Role in TGF-β Actions, ECM Production, and the Diabetic Kidney
- Other Kidney Diseases
- Clinical Applications, Therapeutic Strategies, and Perspectives
- Glossary of Terms
- Disclosures
- Acknowledgments
- References
- Figures & Data Supps
- Info & Metrics
- View PDF
Cited By...
- Gestational low-protein intake enhances the whole kidney miR-192 and miR-200 family expression and epithelial-to-mesenchymal transition in adult male offspring
- Gestational low-protein intake enhances whole-kidney miR-192 and miR-200 family expression and epithelial-to-mesenchymal transition in rat adult male offspring
- Tissue-Specific MicroRNA Expression Patterns in Four Types of Kidney Disease
- Exocytosis-Mediated Urinary Full-Length Megalin Excretion Is Linked With the Pathogenesis of Diabetic Nephropathy
- Aldosterone Regulates MicroRNAs in the Cortical Collecting Duct to Alter Sodium Transport
- FOG2 Protein Down-regulation by Transforming Growth Factor-{beta}1-induced MicroRNA-200b/c Leads to Akt Kinase Activation and Glomerular Mesangial Hypertrophy Related to Diabetic Nephropathy
- MicroRNA-324-3p Promotes Renal Fibrosis and Is a Target of ACE Inhibition
- Analysis of urinary microRNAs in chronic kidney disease
- Translational study of microRNAs and its application in kidney disease and hypertension research
- Abnormal miR-148b Expression Promotes Aberrant Glycosylation of IgA1 in IgA Nephropathy
- Pro-Inflammatory Role of MicroRNA-200 in Vascular Smooth Muscle Cells From Diabetic Mice
- Inhibiting MicroRNA-192 Ameliorates Renal Fibrosis in Diabetic Nephropathy
- The Long and Short of MicroRNAs in the Kidney
- MicroRNA-21 Orchestrates High Glucose-induced Signals to TOR Complex 1, Resulting in Renal Cell Pathology in Diabetes
- Enhanced Levels of microRNA-125b in Vascular Smooth Muscle Cells of Diabetic db/db Mice Lead to Increased Inflammatory Gene Expression by Targeting the Histone Methyltransferase Suv39h1
- Mechanisms of Tubulointerstitial Fibrosis
- Post-transcriptional Up-regulation of Tsc-22 by Ybx1, a Target of miR-216a, Mediates TGF-{beta}-induced Collagen Expression in Kidney Cells
- Targeted Deletion of Dicer from Proximal Tubules Protects against Renal Ischemia-Reperfusion Injury