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Exercise-Associated Hyponatremia

Mitchell H. Rosner and Justin Kirven
CJASN January 2007, 2 (1) 151-161; DOI: https://doi.org/10.2215/CJN.02730806
Mitchell H. Rosner
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Justin Kirven
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    Figure 1.

    Pathophysiologic factors in the development of exercise-associated hyponatremia (EAH). AVP, arginine vasopressin.

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    Figure 2.

    Relationship between serum sodium after racing and the weight change (in %) during exercise in 2135 athletes who competed in endurance events. •, asymptomatic athletes; ○, athletes with symptoms compatible with EAH encephalopathy (EAHE). The majority of athletes who develop clinically significant hyponatremia have positive weight changes. Reprinted from reference (34), with permission. Copyright 2005 National Academy of Sciences.

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    Table 1.

    Risk factors for the development of EAHa

    Exercise duration >4 h or slow running/exercise pace
    Female gender (may be explained by lower body weight)
    Low body weight
    Excessive drinking (>1.5 L/h) during the event
    Pre-exercise overhydration
    Abundant availability of drinking fluids at the event
    Nonsteroidal anti-inflammatory drugs (not all studies)
    Extreme hot or cold environment
    • ↵a EAH, exercise-associated hyponatremia.

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Clinical Journal of the American Society of Nephrology
Vol. 2, Issue 1
January 2007
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Exercise-Associated Hyponatremia
Mitchell H. Rosner, Justin Kirven
CJASN Jan 2007, 2 (1) 151-161; DOI: 10.2215/CJN.02730806

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Exercise-Associated Hyponatremia
Mitchell H. Rosner, Justin Kirven
CJASN Jan 2007, 2 (1) 151-161; DOI: 10.2215/CJN.02730806
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    • Incidence
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